الفهرس 
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Abstract
S
UMMARY
phakic bullous keratopathy is defined as corneal stromal edema and Descemet‘s folds following cataract surgery due to corneal endothelial decompensation and endothelial cell loss with the absence of an IOL.
Penetrating keratoplasty used to be the only treatment choice in such cases, now descemet‘s membrane endothelial keratoplasty (DMEK) has the upper hand in the treatment of bullous keratopathy.
DMEK eliminated the stromal interface aiming for perfect substitution of the endothelial cell layer using descemet‘s membrane as a carrier, allowing perfect anatomical replacement of the diseased endothelium by a healthy donor.
Previous retrospective studies showed the value of DMEK in the treatment of endothelial dysfunction like endothelial dystrophy and pseudophakic bullous keratopathy.
Our study reported very comparable results in treatment of aphakic bullous keratopathy in terms of improved BCVA, CCT and also endothelial cell loss when compared to different techniques of secondary IOL implantation and when compared to a standard DMEK in the treatment of pseudophakic bullous keratopathy or fuchs endothelial corneal dystrophy.
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Summary
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Larger study group and longer follow up periods are needed for longer term assessment of the graft survival and follow up of the visual acuity and the central corneal thickness.