الفهرس 
introduction and aim of the workOnly 14 pages are availabe for public view
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Abstract
Neonatal jaundice constitutes a problem to the newborn if it is beyond the physiologic limit. Neonatal jaundice occurs when the normal pathway of bilirubin metabolism and its excretion in the newborn are impeded, and consequently the pigments are deposited in the skin and mucous membranes producing the characteristic yellowiSh discoloration of these sites. According to our observations, many of full-term babies present during the first week of life with jaundice which is not fulfilling criteria of either pathologic or hemolytic types, yet serum bilirubin level exceeds the physiologic limits, consequently we hypothesized that some factors may predispose these babies to acquire this higher serum bilirubin levels. One presumed factor is tetanustoxoid given to mothers during their pregnancies. In our study, we aimed to uncover the role of tetanus toxoid in the exaggeration of neonatal jaundice in full-term neonates in the first week of life when given to mothers beyond the first trimester. The study included 50 full term newborns with exaggerated unconjugated nonhemolytic hyperbilirubinemia beyond the physiologic limit in the first week of life. They were classified into 2 groups; the first group comprised 38 babies whose mothers had received tetanus toxoid beyond the first trimester and the second .