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العنوان
Pharmacotherapy of renal damage caused by endotoxemia in preeclamptic rats and their offspring/
المؤلف
Morgaan, Hagar Aly Ibrahim Aly .
هيئة الاعداد
باحث / هاجر على ابراهيم على مرجان
مشرف / محمود محمد محمود الماس
مشرف / سحر محمود عزمى الجويلى
مشرف / مروة يوسف محمد سلام
الموضوع
Toxicology
تاريخ النشر
2024.
عدد الصفحات
172 p. :
اللغة
الإنجليزية
الدرجة
ماجستير
التخصص
العلوم الصيدلية
تاريخ الإجازة
1/1/2024
مكان الإجازة
جامعة الاسكندريه - كلية الصيدلة - علم الأدوية والسموم
الفهرس
Only 14 pages are availabe for public view

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from 172

Abstract

Several maternal risk factors have been linked to PE development and may be used to speculate maternal propensity to this condition. These risk factors include nulliparity, short and long interpregnancy interval, previous history of PE or hypertension in pregnancy, advanced maternal age greater than 35 years old, family history of PE, obesity, in-vitro fertilization or use of other forms of assisted reproductive technologies, and other maternal comorbid conditions including pre-existing chronic renal disease, autoimmune disease or hyperglycemia in pregnancy (Poon et al., 2019).
Based on the time of onset of the clinical features, PE can be categorized into two sub-types: early and late onset PE (occurring before or after 34 weeks of gestation) with early-onset PE being more severe and having much greater risk of short- and long-term maternal and perinatal morbidity and mortality (Khodzhaeva et al., 2016; von Dadelszen et al., 2003). Another possible classification of PE is based on the severity of the condition and the degree of morbidity categorizing it into mild or severe PE. Severe features of PE include cases with BP values ≥160/110 mmHg, thrombocytopenia (platelet count less than 100,000/microliter), pulmonary edema