الفهرس | Only 14 pages are availabe for public view |
Abstract nflammation represents a vital defense mechanism that is crucial for maintenance of host health particularly in acute disease states (Abdulkhaleq et al., 2018). However, inflammation becomes an annoyance when the body suffers from unregulated or prolonged inflammation where targeted destruction and intended repair are not appropriately controlled by its mediators leading to persistent host tissue damage that conclude with the insurgence of different pathologies as cardiovascular disease (CVD), atherosclerosis, inflammatory bowel disease (IBD), multiple sclerosis and neurodegenerative diseases (Izano et al., 2016). Several types of synthetic pharmacological therapies are utilized to slow down or suppress the progression of these devastating inflammatory disorders (Perretti et al., 2015). Yet, worst of all, chronic use of such |