الفهرس 
INTRODUCTIONOnly 14 pages are availabe for public view
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Abstract
Neonatal encephalopathy (NE) “as reported by The Task Force on NE group” is defined as a clinical syndrome manifested by malfunctioning of the CNS, in the first days of life in a neonate born at or beyond 37 weeks of gestation, presenting with an altered level of consciousness, epilepsy, abnormal tone and reflexes and usually associated with difficulty in initiating and maintaining respiration.(2) Neonatal encephalopathy can result from a wide variety of conditions; perinatal hypoxic ischemic encephalopathy (HIE) – the most common cause of neonatal encephalopathy- and other non-hypoxic ischemic causes including metabolic disorders from inborn errors of metabolism, neonatal stroke, congenital infections, drug exposure, nervous system malformations and birth trauma.(152)
This study was conducted to evaluate the role of MRI in identification of non-hypoxic ischemic causes of neonatal encephalopathy.
The study was conducted on 50 full term neonates admitted to the neonatal intensive care unit (NICU) at Alexandria University Children’s Hospital (AUCH) with clinical picture suggestive of encephalopathy including; disturbed level of consciousness, feeding difficulties, irritability, abnormality of tone, seizures and difficulties in initiating and maintaining respiration.
MRI examinations were performed on a 1.5 Tesla General Electric (GE) machine with conventional sequences including 3D T1, axial and coronal T2, axial FLAIR, DWI and SWI. MRS was added for cases with suspected metabolic abnormality. MRA and MRV were also done for cases with suspected stroke or vascular causes.
In the current study causes of non -HI