الفهرس 
Anatomy of the RetinaOnly 14 pages are availabe for public view
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Abstract
Diabetic retinopathy is a leading cause of vision loss world wide.
This vision loss is due to leakage of blood constituents from diabetic MAs to the surrounding retinal tissue which causes retinal thickening and macular edema.
The aim of this work was to detect SD-OCT characterizations of diabetic MAs and correlate these findings with the amount of leakage on FA.
This study included 50 diabetic MAs of 50 diabetic patient with diabetic MAs (10 mild NPDR; 21 moderate NPDR; 14 severe NPDR; 5 proliferative diabetic retinopathy PDR).
All patients were submitted to detailed history taking, full ophthalmologic evaluation including FFA and SD-OCT.
Among patients, thirty patients were males while twenty patients were females.
The patients aged between 38.0 – 70.0 years, with a mean of 56.0± 8.21 years.
Among the patient, ten of them {20.0%} had mild NPDR, twenty one {42.0%} had moderate NPDR, fourteen{28.0%}had severe NPDR and five patients{10.0%} had PDR.
Among the fifty diabetic patients, fourteen {28.0%} had type 1 diabetes and thirty sex {72.0%} had type 2 diabetes.
Summary & Conclusions
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Among the patients, 54.0% had diabetes for less than 15 years and 46.0% had diabetes for 15 year or more.
According to leakage, number of microaneurysms with mild angiographic leakage and severe leakage was similar (twenty; 40.0%), while microaneurysms showed no leakage were ten {20.0%}.
External diameter of all microaneurysms ranged between 53.0 – 225.0μm with a mean of 104.02±39.85μm, internal diameter ranged between 28.0 – 131.0 μm with a mean of 56.30±23.52 μm and the thickness of microaneurysms ranged between 10.50 – 62.50 μm with a mean of 22.86 ± 11.20μm.
Percent depth was ranged between 7.0% -54.0 %with a mean of 25.89±10.23%.
The innermost extent ranged from the ganglion cell layer (GCL) to the outer plexiform layer (OPL). The innermost extent of the plurality of microaneurysms (26/50; 52.0%) was the inner plexiform layer (IPL). The outermost extent of microaneurysms ranged from INL to ONL, with the majority of microaneurysms (24/50; 48.0%) having an outermost extent in OPL.
The majority of microaneurysms spanned more than one retinal layer (90.0%), twenty six (26/50, 52.0%) of microaneurysms spanned three retinal layers, fourteen (14/50, 28.0%) spanned two retinal layers, five (5/50,10.0%) spanned four retinal layers and the same percent spanned one retinal layer.
Among the fifty diabetic microaneurysms, twenty seven (54.0%)