الفهرس 
IntroductionOnly 14 pages are availabe for public view
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Abstract
When diagnosing glaucoma or monitoring its progression, ophthalmologists rely on the appearance of the disc, measures of retinal nerve fiber layer thickness and visual fields. However, other disorders of the optic nerve can also produce visual field findings, nerve fiber layer loss and disc appearance that can mimic glaucoma.
Non glaucomatous optic neuropathies(Pseudoglaucoma) have been reported to cause cupping of the optic nerve, mimicking that found in glaucoma .
Pseudoglaucoma is defined as false or deceptive glaucoma with or without ocular damage. Multiple entities can produce ”pseudoglaucomatous optic disc” and should have a special attention to differentiate from glaucomatous eyes that behave, at least for a time, as though it did not have glaucoma . They include the following :
a physiological or congenital large optic cup .congenital anomalies of optic nerve , like congenital pits , colobomas , the morning glory syndrome and the tilted optic disc . pale, cupped nerve can sometimes result from diseases such as ischemic optic neuropathy , optic neuritis, toxic optic neuropathy , shock optic neuropathy , compressive lesions , leber’s hereditary optic neuropathy and dominant optic atrophy .
Visual field and OCT appears to be a useful technology in evaluation pseudoglaucomatous optic disc cupping . The pattern of RNFL loss was varied depending upon the etiology and appears more diffuse in non-glaucomatous optic nerve cupping .The nasal and temporal RNFL thickness were lower in patients with non- glaucomatous optic nerve cupping compared to the reported results in glaucoma. The pattern of visual field defect was varied depending on the etiology and had better visual field MD and PSD compared to reported results in glaucoma.