الفهرس 
INTRODUCTIONOnly 14 pages are availabe for public view
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Abstract
IN ’l’ RODUCTION
Haemolytic disorders have been subjected to intensive
studies in the last few years • Many new mechanisms and
metabolic pathways involved in either erythropoiesis or
red cell destruction were put to light as a result of
these studies ( Wintrobe, 1981 ) •
:Many factors were blamed for introduction o f intravascular
haemolysis as heriditary factors, immunological
factors, chemicals, drugs, the traumatic factors • • • etc.
G6PD deficiency is a common disease with different
v.arieties ( Yo.shida, 1973 ) • The effect of diabetic ketoacidosis
in inducing haemolytic disease in the patients
with G-6-P D deficiency was heavily investigated (Gellady,
1972 ) •
The role of excessive lysis in acidified human serum
is one of the char>Jcteristic features of PNII cells •
Many metabolic and r:1echan.ical factors we r e inve::r tignted
and discussed as regards their effect on the haemolytic
disorders . The effect of hyperglycaemia with or without
ketoacidosis were investigated as regards their injurious
effect on the R.B.Cs.
G-6-P D = Glucose -6- phosphate dehydrogenase
PNH = Paroxysmal nocturnal haemoglobinuria •