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Abstract Diabetes mellitus is one of the commonest endocrinal diseases in childhood. It is classified into three forms, type I diabetes, type II diabetes and secondary diabetes. The basic cause of type I diabetes in childhood is the sharply diminished secretion of insulin. (Genuth. 1983). Although basal insulin concentrations in plasma may be normal in newly diagnosed patients insulin production in response to a variety of potent secretagogues is blunted and usually disappears over a period of months. to years rarely exceeding 5 years. (Sperling, 1988). Type I diabetes is characterized by increased frequency of complications (Brittle diabetes). Among the important complications are the neurological complications. (Feingold. 1984). EEG abnormalities constitute one of the neurological complications which are suspected to occur during the course of type I diabetes mellitus. The hypoglycemia and ketoacidosis which occur in the type I diabetes are the 1 major factors incriminated in the occurrence of the EEG changes. These EEG changes may vary between simple transient forms to severe and permanent EEG sequelae. (Lerman et al., 1977). 2 |