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Abstract Introduction and Aim of the Work --· I Introduction and Aim of the Work Artificially led infants gain weight more rapid than breast fed int3nts and this reflects the effect of overfeeding in artificially fed infants . Such overfeeding might contribute to subsequent obesity because of developement of an abnormally large population of adipocytes or because of establishment of a habit of overfeeding. The diet of infants depending on artificial feeding contain high fat content which lead to delay gastric emptying ,causing distension , abdominal discomfort and excessive gain in weight. (Joseph eta! , 1970) In breast fed infants the intake of calories is less than of bottle fed infants and gain of weight has been demonstrated to be significantly related to caloric intake ( Fornon, 1972) Leptin , a product of obesity gene , is a hormone that is secreted into the blood in varying quantities by adipocytes and control the adipose tissue weight by stimulating lipid metabolism ( Pankov, 1996) It was found that there is strong relation hetween leptin and obesity also leptin is increased in adipose tissue in response to feeding.( Friedman et al, 1996) As far as could be ascertained, this is the first pilot study regarding the assessment of serum lcptin in relation to feeding whether breast or artificial feeding |