الفهرس | Only 14 pages are availabe for public view |
Abstract The present study was performed to investigate the clinicopathological changes associated with acute and chronic heat exposure in chickens. One hundred one day old Hubbard chicks were used for this experiment. They divided into five equal groups. The first gp. kept as negative control (non treated non heat stressed), gp. (2) were exposed to 42±l QC for 3 hrs. with RH. 45- 60% at 45 days old, gp. (3) were treated with Anti heat stressl gmIliter water orally 24hrs. before heat exposure and exposed to 42±1 QC for 3hrs with RH. 45-60%. at 45 days old, gp. (4) were exposed to 39±1 QC for 6 hrs. for 3 successive days with RH. 45- 60%. at 43, 44 and45 days old while gp. (5) were treated with Anti heat stressl gmlliter water orally 24hrs. before heat exposure and exposed to 39±1 QC for 6 hrs. for 3 successive days with RH. 45- 60%. at 43, 44 and 45 days old. Clinical signs was observed. Blood and serum samples were collected at the same time (0, 12 and 36 hrs. after heat exposure) for hematological and clinicobiochemical studies. Tissue specimen from liver, kidneys, lung and heart from all gps. were examined macroscopically and microscopically. Chickens exposed to acute and chronic heat exposure (gps. 2 and 4) were standing with wings outstretching, off food, panting (open mouth breathing) and laying on the ground during periods of exposure with mortality rate reached 20% in acute exposure. Gps. (3 and 5) exposed to acute and chronic heat exposure and treated with no-heat stress) showed some improvement in the clinical signs. |