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Abstract The results of this study show that FMD (reactive hyperemia sponse) was significantly decreased in the ED group compared with the :ontrol group (4.08±1.7% vs 10.4±1.4%, P <0.001) indicating impaired ascular response to reactive hyperemia in patients with ED. Non :ndothelial vasodit~tion (GTN dilation%) was lower but statistically nsignificant in patients with ED than in control subjects (l3.8±2.1% vs 4.4±2.2%, P =0.32 not significant). The relationship between ED and , 0 was significant (r=0.87, P< 0.001) (Figure 2), whereas no lationship was found between ED and NDD (r =0.13, P <0.32). The SBP, DBP, PP and AoD did not differ significantly between ~oups (Table 3). Aortic strain (3.3±0.95% vs 10.09±2.2%, P<O.OOI) and listensibility (1.9±0.64 vs 4.7±1.1 cm2 x dyne-l xl0-6, P <0.001) were ound significantly lower in the ED group than in the control group while \ortic stiffness index (3.9±0.51 vs 2.8±0.2, p<O.OOI)was found lignificantly higher in the ED group than in the control group. The relationship between ED and aortic stiffness was also significant (for ortic strain, r=0.81, P <0.001; for aortic distensibility, r=0.78, P <0.001; md for ASI, r= -0.76, p<O.OOI) . |