الفهرس 
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Abstract
Asherman’s syndrome is defined by the presence of permanent
intrauterine adhesions (IUA), formed during the scarring of endometrial
surface defects, and obliterating partially or completely the uterine cavity.
The presence of such adhesions has been known to be associated with
hypomenorrhea or amenorrhea, infertility, and obstetric complications.
The most frequent causes of their formation are postpartum or
postabortion overzealous dilatation and curettage. Other uterine infections
or surgical trauma might also be the predisposing factor (Asherman,
1948, Rabau et al., 1963; Forssman, 1965; Carmichael, 1970;;
Schenker et al., 1982; Kurman et al., 1994; and Pabuçcu et al., 1997).
Surgical excision of the intrauterine adhesions has evolved over time.
In the past, access to the uterine cavity was by direct abdominal approach
using laparotomy or via the transcervical route with blind division of
adhesions using a uterine sound. This did not allow analyzing results in
relation to proper visual assessment of the severity of the adhesions and
the extent of uterine cavity occlusion. The use of hysteroscopy in such
case since the early 1970s, particular with the newer instrumentation that
allows for easier and faster dissection, has allowed rapid improvement in
the diagnosis, assessment, and treatment of IUA (March et al.,1978;
Sanfilippo et al.,1982; Otubu et al., 1989; Dicker et al., 1996; and
Amer et al., 2005).