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Abstract
Alherosclerosis, which involves the formation of fatty deposits or plaques in the inner wall of arteries, is now considered in many ways an inflammatory disorder of the blood vessels.
• Inflammation not only affects the alherosclerotic phase of heart disease, but also the rupturing of plaques which can then travel and interfere with blood flow, causing acute myocardial infarction.
• Many inflammatory markers are involved in the mechanism of plaque formation and rupture. One of these inflammatory markers, C. reactive pretein (CRP), is shown to have prognostic significance, in both short and long term in acute myocardial infarction. Additionally, it has been suggested that elevated CRP levels after acute myocardial infarction is correlated with infarct size, and has predictive value for post-MI complications such as left ventricular failure or cardiac rupture.